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| OverviewBasic ScienceTranslational ResearchClinical ResearchIslet TransplantationPublications ListTraining OpportunitiesDiabetes Research LinksThe Hormone Research InstituteThe Metabolic Research Unit | Fred Schaufele, Ph.D Schaufele F, Wang X, Liu X, Day RN 2003 Conformation of CCAAT/enhancer binding protein alpha dimers varies with intranuclear location in living cells. J Biol Chem: 278: 10578-10587. Enwright III JF, Kawecki-Crook MA, Voss TC, Schaufele F, Day RN 2003 A Pit-1 homeodomain mutant blocks the intranuclear recruitment of the CCAAT/enhancer binding protein alpha required for prolactin gene transcription. Mol Endocrinol 17: 209-222. Day RN, Voss TC, Enwright III JF, Booker CF, Periasamy A, Schaufele F 2003 Imaging the localized protein interactions between Pit-1 and the CCAAT/enhancer binding protein alpha in the living pituitary cell nucleus. Mol Endocrinol 17: 333-345. Weatherman RV, Chang C-Y, Clegg NC, Carroll DC, Day RN, Baxter JD, McDonnell DP, Scanlan T, Schaufele F 2002 Ligand-dependent interactions of estrogen receptor detected in vivo by Fluorescence Resonance Energy Transfer. Mol Endocrinol 16: 487-496. Liu W, Enwright JF, Hyun W, Day RN, Schaufele F 2002 CCAAT/Enhancer Binding Protein alpha uses distinct domains to prolong pituitary cells in the Growth 1 and DNA Synthesis phases of the cell cycle. BMC Cell Biology 3: 6, pp. 1-11. online journal, http://www.biomedcentral.com/1471-2121/3/6 | Related Items |